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Welcome
The Limousin Muscle Alliance was founded in 2009 by a group of like-minded Limousin breeders and enthusiasts who are concerned about the erosion of muscling and other traditional breed standard values for which Limousin cattle are renowned.
These values of calving ease, carcase yield and efficiency have stood the test of time and are the reason for Limousin's phenomenal rise in popularity as the beef sire breed of choice in many countries throughout the world.
Limousin cattle originated in France and research since the mid 1990s has shown that French Pure cattle carry two copies of a muscling gene (Myostatin variant F94L) which is unique to the breed. In the grading up process to produce Australian Purebred Limousins, the presence of this gene has been maintained although its prevalence has been slightly reduced in the grading up process with some cattle only carrying one or zero copies of the gene.
In order to consistently pass on at least one F94L gene and produce superior muscling and shape in progeny, the Limousin sire must carry 2 copies of the F94L gene. These are the sires that can truly boast "carcase, yield, efficiency" benefits. Commercial breeders utilising both male and female purebred Limousin cattle, carrying two copies of the "muscle gene" are gaining the full benefits of "19% more beef for the same amount feed" from the Limousin muscle gene.
Alliance members breed both French Pure and Australian Pure cattle and are dedicated to genetic progress without losing Limousin's original prepotent characteristics. They are committed to the production of Limousin cattle which will derive the full benefits of the muscle gene for their clients. To source Limousin "muscle gene" genetics or further information on the benefits of "muscle gene" cattle please feel free to contact one of our members.
FOUNDING MEMBERS
Keystone Genetics, Benamitchy Limousin, Lakella Limousin, Tanholm Limousin
Photos of Bulls and Females used throughout this site are either French Pure or Pure high content Limousin tested to carry the F94L gene.
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